Complement C5a 靶点蛋白|抗体药物|临床药物信息

Complement C5a信息

英文名称：Complement C5a
中文名称：补体C5a
靶点别称：C5a Complement，Complement 5a，Complement Component 5a

上市药物数量：0
临床药物数量：0
最高研发阶段：终止

Complement C5a产品列表

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在线 (6)

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属性

Protein (6)

标记

Unconjugated (6)

货号	物种	产品描述
C5A-H5116	Human	Human Complement C5a Protein, Tag Free
C5A-M51H4	Mouse	Mouse Complement C5a Protein, His Tag
C5A-C5118	Cynomolgus	Cynomolgus Complement C5a Protein, Tag Free
C5A-H525a	Human	Human Complement C5a Protein, Fc Tag (MALS verified)
C5A-C51H9	Cynomolgus	Cynomolgus Complement C5a Protein, His Tag
C5A-H51H9	Human	Human Complement C5a Protein, His Tag

C5A-H5116|Human Complement C5a Protein, Tag Free

176XXXXXXX3

2人赞

因为实验室用的蛋白种类比较多，所以Acro，Sino，R&D的产品都有在用，因为Acro到货最快，所以一直都是我们的第一选择，产品质量也很赞。但有时候做实验也会发现不同厂家的产品会有差异，同样的氨基酸序列，同样的浓度，很想咨询一下这种差异来源是什么呢？每次实验前买好几种产品真的很麻烦￣□￣｜｜
2020-07-01

C5A-H5116|Human Complement C5a Protein, Tag Free

151XXXXXXX5

2人赞

从Acro订购太多蛋白了，这一款给我印象深刻，不说试用装很大方地送我做预实验，我所需的蛋白他们还会开发不同种属的，节省了我很多时间，最重要的，相关蛋白，他家不止做结合实验，还会把相关活性实验也进行检测，而且还分享给客户，那是相当周全了。愿所有的蛋白你家都有。
2019-09-26

Complement C5a 分子别名

C5a,Complement Component 5a

Complement C5a 分子背景

Complement Component C5a (C5a) is also known as C5,and is a protein fragment released from complement component C5.,is a potent chemotactic factor for human peripheral blood neutrophils and monocytes, and is believed to play an important role in a number of inflammatory conditions. There are several functions in the below: C5a is an anaphylatoxin, causing the release of histamine from mast cells; C5a is effective leukocyte chemoattractants, causing the accumulation of white blood cells, especially neutrophil granulocytes, at sites of complement activation; C5a activates white blood cells by increasing avidity for white blood cell integrins and upregulating the Lipoxygenase pathway for arachidonic acid metabolism; C5a is a powerful inflammatory mediator, and seems to be a key factor in the development of pathology of many inflammatory diseases involving the complement system; C5a modulates balance between activating versus inhibitory IgG Fc receptors on leukocytes, thereby enhancing autoimmune response.

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